“Those who do not find time for exercise will have to find time for illness.”
Our lifestyle is paramount to our health and well-being. We, as a society, have gotten away from the environment that best suits our body. We were designed to live outdoors with fresh air and sunlight. We were designed to roam and walk this earth hunting and gathering food for our survival. Instead, we have created a mostly sedentary lifestyle with today's industrialization. We have become like wild animals in captivity. We are mostly indoors where the air quality is 7 times worse than outside air (even though its polluted out there as well). We don't get sun exposure and we have been convinced that if we dare being in the sun we should first slather on toxic sunblock. This might be why people are so vitamin D deficient. Vitamine D deficient people are 11X higher risk of being depressed than those who have adequate amounts of Vitamin D. Do yourself a favor and look up what other problems are associated with Vitamin D deficiency. Better yet, ask the Drs. Kimberlin. Back to the wild animal comparison, when wild animals are put in captivity they have increased rates of cancer, obesity, heart disease and infertility. Sound like anyone you know?
When you watch shows like The Biggest Loser you see people working very hard to change their habits hopefully transforming them into lean, strong, healthier and happier people. We submit that if they were doing what was more congruent all along, essentially creating an environment more appropriate for their genes/genome, they would not have become morbidly obese. In other words, by not living consistent with the human species in the first place the effects are hormone imbalance, weight gain and health decline. Eating well, moving well and thinking well will improve all of our lives thus making for a Far Better Life!!
Sedentary living and immobility. People generally don't move enough and certainly don't move with a high intensity which comes with a full throttle dash like if you were chasing after your dinner. Instead we sit around eating dinner that has been chemically processed and preserved adding to the mounting level of chemicals in our bodies. Flame retardent mattresses, detergents, beauty products, pesticides, and about 80,000 other man made chemicals we now have in our environment (very few have been regulated by the Federal Govt). Emotional distress is at an all-time high, at least in our lifetime, with 2 wars, an economic meltdown, +10% unemployment, an oil spill in the gulf (releasing over 80 million gallons) ... all this with no end in sight. It's safe to say we all have felt a little more stress on top of simply running a household. We will always have some form of stress or another. We either deal with it constructively and process it, or, we don't and that stress is internalized and negativley effects our health. Hans Selye's work proves that what we think, can and does make us sick. For some, it can lead to their demise. These are all examples of how Vertebral Subluxations are created. Movement does a lot to first prevent and secondley help the body self correct Vertebral Subluxation.
Research shows that exercise improves Heart Rate Variability (HRV).
Our Heart Rate Variability (HRV) test is a great way to measure the effects of stress on our overall health.
HRV is a reliable overall health measurement because when diminished it is associated with the following.
Increased mortality risk
Increased risk for arrhythmic events
Depressed patients with coronary artery disease
Elevated HRV is associated with the following: (Increases HRV...a good thing)
Chiropractic adjustments due to the shift to a healthy autonomic Nervous System balance.
Want to read what the scientists say about HRV?
Heart rate variability (HRV) refers to the beat-to-beat alterations in heart rate. Under resting conditions, the ECG of healthy individuals exhibits periodic variation in R-R intervals. This rhythmic phenomenon, known as respiratory sinus arrhythmia (RSA), fluctuates with the phase of respiration -- cardio-acceleration during inspiration, and cardio-deceleration during expiration. RSA is predominantly mediated by respiratory gating of parasymphathetic efferent activity to the heart: vagal efferent traffic to the sinus node occurs primarily in phase with expiration and is absent or attenuated during inspiration. Atropine abolishes RSA.
Reduced HRV has thus been used as a marker of reduced vagal activity. However, because HRV is a cardiac measure derived from the ECG, it is not possible to distinguish reduced central vagal activity (in the vagal centers of the brain) from reduced peripheral activity (the contribution of the target organ -- the sinus node -- or the afferent/efferent pathways conducting the neural impulses to/from the brain).
What aspect of allostasis does HRV potentially measure? ("allostasis" or "maintaining stability through change",)
Although our understanding of the meaning of HRV is far from complete, it seems to be a marker of both dynamic and cumulative load. As a dynamic marker of load, HRV appears to be sensitive and responsive to acute stress. Under laboratory conditions, mental load -- including making complex decisions, and public speech tasks -- have been shown to lower HRV. As a marker of cumulative wear and tear, HRV has also been shown to decline with the aging process. Although resting heart rate does not change significantly with advancing age, there is a decline in HRV, which has been attributed to a decrease in efferent vagal tone and reduced beta-adrenergic responsiveness. By contrast, regular physical activity (which slows down the aging process) has been shown to raise HRV, presumably by increasing vagal tone.
In short, HRV appears to be a marker of two processes, relevant to the conceptualization of allostatic load: (1) frequent activation (short term dips in HRV in response to acute stress); and (b) inadequate response (long-term vagal withdrawal, resulting in the over-activity of the counter-regulatory system -- in this case, the sympathetic control of cardiac rhythm).
The major reason for the interest in measuring HRV stems from its ability to predict survival after heart attack. Over half a dozen prospective studies have shown that reduced HRV predicts sudden death in patients with MI, independent of other prognostic indicators such as ejection fraction. Reduced HRV appears to be a marker of fatal ventricular arrhythmia. Moreover, a small number of studies have begun to suggest that reduced HRV may predict risk of survival even among individuals free of CHD.
Copyright © 1999 UCSF
Contact: Judith Stewart
Revised 2 June 2000
The term allostatic load, coined by McEwen and Stellar in 1993, is defined as the physiological consequences of chronic exposure to fluctuating or heightened neural or neuroendocrine response that results from repeated or chronic stress. It is used to explain how frequent activation of the body's stress response, essential for managing acute threats, can in fact damage the body in the long run. Allostatic load is generally measured through a composite index of indicators of cumulative strain on several organs and tissues, but especially on the cardiovascular system.
The hormones and other physiological agents that mediate the effects of stress on the body have protective and adaptive effects in the short run and yet can accelerate pathophysiology when they are over-produced or mismanaged.
Adaptation in the face of stressful situations and stimuli involves activation of neural, neuroendocrine and neuroendocrine-immune mechanisms. This adaptation has been called "allostasis" or "maintaining stability through change", which is an essential component of maintaining homeostasis. The main hormonal mediators of the stress response, cortisol and epinephrine (adrenaline), have both protective and damaging effects on the body. In the short run, they are essential for adaptation, maintenance of homeostasis, and survival “allostasis”. Yet, over longer time intervals, when called upon frequently, they exact a cost “allostatic load” that can accelerate disease processes. Allostatic load can be measured in physiological systems as chemical imbalances in autonomic nervous system, central nervous system, neuroendocrine, and immune system activity as well as perturbations in the diurnal rhythms, and, in some cases, plasticity changes to brain structures.
Four conditions that lead to allostatic load are:
- Repeated frequency of stress responses to multiple novel stressors;
- Failure to habituate to repeated stressors of the same kind;
- Failure to turn off each stress response in a timely manner due to delayed shut down; and
- Inadequate response that leads to compensatory hyperactivity of other mediators.
The effects of these forms of dysfunctional allostasis cause allostatic load and this, over time, leads to diseases. Allostatic load effects can be measured in the body. When tabulated in the form of allostatic load indices using sophisticated analytical methods, it gives an indication of cumulative lifetime effects of all types of stress on the body.
- ^ Taylor, S. E. (2006). Health Psychology. McGraw-Hill Education, pg. 160
- ^ a b McEwen B. S. (2000). "Allostasis and allostatic load: implications for neuropsychopharmacology". Neuropsychopharmacology 22 (2): 108–24. doi:10.1016/S0893-133X(99)00129-3. PMID 10649824.
The Pulse Wave Profiler is our HRV device, see it at the computerized health assesment section.